Project DAAD GradSchool-Prgm – Role of Akt signaling in homologous recombination-dependent repair of radiation-induced DNA…

Basic data

Acronym:
DAAD GradSchool-Prgm
Title:
Role of Akt signaling in homologous recombination-dependent repair of radiation-induced DNA double strand breaks
Duration:
01/10/2016 to 30/09/2019
Abstract / short description:
Akt is a transducer kinase which acts as a signaling component for cell survival and DNA repair. On the basis of previous results indicating a defined role of Akt in regulating DNA double-strand breaks through the non-homologous end-joining mechanism, the current project will analyse the potential influence and regulatory function of Akt in the control of the homologous recombination process, which is the second mechanism for DNA-double-strand breaks in eukaryotic cells.
Keywords:
Akt und DNA-Reparatur

Involved staff

Managers

Faculty of Medicine
University of Tübingen

Contact persons

Faculty of Medicine
University of Tübingen

Local organizational units

Department of Radiation Oncology
Hospitals and clinical institutes
Faculty of Medicine

Funders

Bonn, Nordrhein-Westfalen, Germany
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